This study uncovers that activation of endogenous retroviruses affects prion-like spreading of proteopathic seeds. It supports that endogenous retroviruses play a role in protein misfolding diseases and suggest that antiviral drugs could represent promising candidates for inhibiting protein aggregate spreading.
https://www.nature.com/articles/s41467-023-40632-z (open access)
You must log in or register to comment.
Interesting. Does this mean Alzheimer’s might come from an ERV?
At least the ERVs seem to play a role in the progression of the disease.
Removed by mod
Removed by mod
Removed by mod
Removed by mod
Removed by mod
Removed by mod
Removed by mod
Removed by mod
Removed by mod
Removed by mod
Removed by mod
Removed by mod
Removed by mod
Removed by mod